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Correct
As illustrated
with the data shown here, the sensation of effort or work of breathing
was reduced by the institution of mechanical ventilation and the
alleviation of the load on the inspiratory muscles, but the sensation
of chest tightness was not altered. These data support the notion
that chest tightness does not arise from activity of the ventilatory
muscles and are consistent with the hypothesis that stimulation
of pulmonary receptors by bronchospasm may play a critical role
in the origins of this type of dyspnea. Studies of bronchoconstriction
in patients with high cervical spinal cord injury and absent sensory
information from the chest wall and diaphragm may provide even
more compelling information in support of this hypothesis (Cristiano
et al., 1994).
| Figure
11.1: Sensations of Tightness (Left Panel) and Effort
to Breathe(Right Panel) Made During Mechanical and Spontaneous
Ventilation with the Highest Level of Bronchoconstriction
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Offical
Journal of the American Thoracic Society. © American
Thoracic Society.
Reprinted with permission. |
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